   Most Popular Categories
 Unlimited poweR of GenetiC
EngineerinG
Inventor’s
mouth.
Consequences of my research.
Introduction.
Mysteries in
genetic sciences.
What is
coronary artery disease?
Misconceptions in cardio-vascular
sciences.
Foundation
on which building of Baruah Applied Human.
Revolution in medical sciences rocked by
Dr.D.R. Baruah,FRCSGlas.
Unfolding the
mysteries in human genetic sciences.
Mysteries in
human genetic sciences.
How does the
mutation expresses in particular disease form?
Selection of
patients for gene analysis
Eradication of
heart disease & rarest of the rare diseases- Human genetic studies
through sequencing of m-RNA.
Signal
Transduction plays a major role during pre-bypass and post-bypass
events.
How bypass
surgery triggers signal transduction & phenotypically expressed.
Mutation
Selection of
genes causing heart & other diseases.
Hypoxia,
reactive oxygen species, intracellular calcium & Baruah syndrome.
Re-sequencing
of the following genes to identify the mysteries.
First time on
this planet– Genovac.
Baruah applied
human genetic engineering- a choice of treatment for Cancer.
TGA-A New
Method of Treatment of Complex Congenital Heart Disease.
Endocardial Cushion defect.
Genetic
Engineering–To cure the rarest of the rare autoimmune.
First time on
the Planet–Manifestation of Baruah Syndrome–Moyamoya
The rarest of
the rare genetic disorder–Takayasu.
Isolated
congenital Right Ventricular Hypertrophy.
---------------------------------------
-----------------------
Applied Human Genetic Engineering - Vol.II |
|
Selection of
patients for gene analysiS
We have treated 1500 cases of
coronary artery disease by using Baruah Applied human
genetic engineering with 100% success. These studies
have been carried out since last six and half years and
the patients were grouped in following manner.
isolated CAD with partial
blockages of peripheral arteries
patients with post bypass
surgery associated with stenting, high blood
pressure, type I diabetes mellitus, gout. (one case
of this group had permanent pacemaker implantation
because of inadequate ventricular performance after
bypass surgery)
This group of patients were
having triple vessel disease with diabetes and
nephropathy.
We had selected 10 patients of each
group and 6 healthy non-diseased volunteers selected as
controls with age between 25-35, three females & 3males
each. The genes were analysed from the control group and
found to be healthy and non-mutated. Against this
control group, the first group of 10, 4 females and 6
males aged between 40-55.
Second group of patients were 5
females and 5 males each were showing mutation in genes.
First group was injected Baruah
biological molecules and 24 hours later, mutations from
genes were studied.
Duration of experiment was 2 months,
genes were decoded using Baruah biological molecules.
Third group-After injecting
Baruah biological molecules, clinically signs and
symptoms disappeared and gene expression become
permanent and irreversible.and their genetic analysis
was analysed.
 The following genes are identified
and studied :
 Homo sapiens endoplasmic reticulum golgi
intermediate compartment protein(ERGIC), transcript
variant 2, mRNA.-
Length 818 bases; Molecular Weight 390037.6D
Homo sapiens 1-acylglycerol-3-phosphate
O-acyltransferase 1(lysophosphatidic acid
acyltransferase, alpha) (AGPAT1), transcript variant
1, mRNA.
Length 2242 bases; Molecular Weight 689480D
Homo sapiens coatomer protein complex,
subunit zeta 1 (COPZ1), mRNA.
Length 1900 bases; Molecular Weight 586102.8D
Homo sapiens G-protein signaling modulator
3 (AGS3-like, C. elegans) (GPSM3), mRNA.
Length 1472 bases; Molecular Weight 453407.6D
Homo sapiens Notch homolog 4 (Drosophila)
(NOTCH4), mRNA.
Length 6762 bases; Molecular Weight 2086456.4D
|
Copyright© DR Dhani Ram Baruah Heart City2007
Best view in 1024 by 768 pixels |
| | | |
